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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PLCβ2 Promotes VEGF-Induced Vascular Permeability
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
IJMS, Free Full-Text
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Vascular Endothelial-Cadherin Stabilizes at Cell–Cell Junctions by
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Protein Interactions at Endothelial Junctions and Signaling
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate promotes macrophage HMGB1 lactylation, acetylation, and
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